Which medication can cause increased aqueous humor production and may be a concern for patients with glaucoma?

The medication that can cause increased aqueous humor production and may be a concern for patients with glaucoma is indeed the beta-2 adrenergic agonists. These agents are known to stimulate adrenergic receptors, which can lead to various physiological effects including the potential for increased aqueous humor production in the eye. This increase can exacerbate conditions like glaucoma, where managing intraocular pressure is crucial to prevent optic nerve damage.

In glaucoma management, the goal is often to reduce aqueous humor production or enhance its outflow to mitigate elevated intraocular pressure. Therefore, any medication that can potentially increase aqueous humor production needs to be used with caution in these patients.

Other medications mentioned do not have the same effect on aqueous humor production. For instance, ipratropium is an anticholinergic used primarily for respiratory purposes and does not significantly affect aqueous humor dynamics. Dexamethasone is a corticosteroid that can have various ocular effects but primarily focuses on reducing inflammation rather than increasing fluid production. Latanoprost is a prostaglandin analog specifically used to lower intraocular pressure by increasing aqueous humor outflow, making it an option for glaucoma treatment rather than a concern.

Thus, the involvement of beta-2 adrenergic agonists in potentially raising